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Overexpression of Igf2-derived Mir483 Inhibits Igf1 Expression and Leads to Developmental Growth Restriction and Metabolic Dysfunction in Mice

Ionel Sandovici,Denise S. Fernandez-Twinn, Niamh Campbell,Wendy N. Cooper,Yoichi Sekita,Ilona Zvetkova,David Ferland-McCollough,Haydn M. Prosser, Lila M. Oyama,Lucas C. Pantaleao,Danilo Cimadomo,Karina Barbosa de Queiroz, Cecilia S. K. Cheuk,Nicola M. Smith,Richard G. Kay,Robin Antrobus,Katharina Hoelle,Marcella K. L. Ma,Noel H. Smith, Stefan H. Geyer,Lukas F. Reissig,Wolfgang J. Weninger,Kenneth Siddle,Anne E. Willis,Brian Y. H. Lam,Martin Bushell,Susan E. Ozanne,Miguel Constancia

Cell reports（2024）

Cited 0|Views6
Key words
genomic imprinting,microRNAs,miR-483,insulin-like growth factor 2,IGF2,insulin-like growth factor 1,IGF1,growth hormone,GH,mid-gestation lethality,fetal growth restriction,adiposity,metabolism
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