Inhibition of Glycogen Synthase Kinase-3 Ameliorates β-Amyloid Pathology and Restores Lysosomal Acidification and Mammalian Target of Rapamycin Activity in the Alzheimer Disease Mouse Model: IN VIVO AND IN VITRO STUDIES
Journal of Biological Chemistry（2013）
关键词
Alzheimer Disease,Amyloid,Cell Signaling,Drug Action,Glycogen Synthase Kinase 3,Lysosomal Acidification,Neurodegenerative Diseases,Presenilin,S6 Kinase,mTOR
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