C1ql4 regulates breast cancer cell stemness and epithelial-mesenchymal transition through PI3K/AKT/NF-κB signaling pathway.
Frontiers in oncology(2023)
摘要
Our findings suggest that C1ql4 promotes the BC cell stemness and EMT modulating the PI3K/AKT/NF-κB signaling, and provides a promising target for BC treatment.
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关键词
breast cancer cell stemness,breast cancer,pathway,epithelial-mesenchymal
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