Multifactorial white matter damage in the acute phase and pre-existing conditions may drive cognitive dysfunction after SARS-CoV-2 infection-Neuropathology-based evidence

E. Gelpi, S. Kotz,M. Beyerle,S. Wischnewski,V. Harter, H. Kirschner, K. Stolz,C. Reisinger, E. Lindeck-Pozza,A. Zoufaly,M. Leoni,G. Gorkiewicz,M. Zacharias,C. Haberler,J. Hainfellner,A. Woehrer,S. Hametner,T. Roetzer, T. Voigtlaender,G. Ricken, V. Endmayr,C. Haider, S. Lang,I. Simonitsch-Klupp,C. Kornauth,M. Nackenhorst, J. Klaeger,R. Kain, A. Chott, R. Wasicki,R. Krause,G. Weiss, J. Loeffler-Rag,T. Berger, P. Moser, A. Soleimann,M. Asslaber,R. Sedivy,N. Klupp,M. Klimpfinger,D. Risser,H. Budka,L. Schirmer,A. K. Probstel, R. Hoetberger

Viruses（2023）

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摘要

Our results support some previous neuropathological findings of apparently multifactorial and most likely indirect brain damage in the context of SARS-CoV-2 infection rather than virus-specific damage, and they are in line with the recent experimental data on SARS-CoV-2-related diffuse white matter damage, microglial activation, and cytokine release.

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关键词

COVID-19,SARS-CoV-2,leukoencephalopathy,neuropathology,white matter

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