Physiological responses to acute hypoxia in the liver of largemouth bass by alteration of mitochondrial function and Ca2+ exchange

Liu Lan Zhao,Lei Liao,Hao Xiao Yan,Xiao Hong Tang,Kuo He, Qiao Liu,Jie Luo,Zong Jun Du,Shi Yi Chen,Xin Zhang,Zhang Cheng,Song Yang

Aquatic toxicology (Amsterdam, Netherlands)（2023）

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摘要

•Hypoxia triggered ROS production by regulating ETC activity.•Hypoxia changed mitochondrial morphology by mitochondrial fusion, fission and biosynthesis.•Hypoxia regulated nutrient utilization by inducing autophagy and modulating TCA cycle.•Hypoxia disrupted Ca2+ homeostasis and activated ER quality control.

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关键词

Ca(2+) homeostasis,ER,Hypoxia exposure,Largemouth bass,Mitochondria

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