Kainic acid induced hyperexcitability in thalamic reticular nucleus that initiates an inflammatory response through the HMGB1/TLR4 pathway.

Neurotoxicology（2023）

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摘要

KA induced hyperexcitability activates the HMGB1/TLR4 pathway, which potentially leading to neuroinflammation in epilepsy.

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关键词

AMPA,Epilepsy,HMGB1/TLR4 signaling pathway,Slice patch clamp,Thalamic reticular nucleus

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