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JAK-STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States.

Laura E Stevens,Guillermo Peluffo,Xintao Qiu,Daniel Temko,Anne Fassl,Zheqi Li,Anne Trinh,Marco Seehawer,Bojana Jovanović,Maša Alečković, Callahan M Wilde,Renee C Geck,Shaokun Shu,Natalie L Kingston,Nicholas W Harper,Vanessa Almendro, Alanna L Pyke,Shawn B Egri,Malvina Papanastasiou,Kendell Clement,Ningxuan Zhou,Sarah Walker, Jacqueline Salas,So Yeon Park,David A Frank,Alexander Meissner,Jacob D Jaffe,Piotr Sicinski,Alex Toker,Franziska Michor,Henry W Long,Beth A Overmoyer,Kornelia Polyak

Cancer research（2023）

引用 11|浏览45
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摘要
Chemotherapy resistance in inflammatory breast cancer is driven by the JAK2/STAT3 pathway, in part via cAMP/PKA signaling and a cell state switch, which can be overcome using paclitaxel combined with JAK2 inhibitors.
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关键词
inflammatory breast cancer,jak–stat,breast cancer,chemotherapy-resistant
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