NMNAT2 Inhibition Aggravates Neurological Damage after Traumatic Brain Injury in a Rat Model.

Xiaoyu Gu,Haibo Ni,XuGang Kan,Chen Chen,Zhiping Zhou, Zheng Ding,Di Li,Bofei Liu

Journal of Korean Neurosurgical Society（2022）

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摘要

NMNAT2 expression is increased and NMNAT2 exhibits neuroprotective activity in the early stages after TBI, and Bax signaling pathway may be involved in the process. Thus, NMNAT2 is likely to be an important target to prevent secondary damage following TBI.

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关键词

Bax,NMNAT2,Neurological deficit,Neuronal degenerative,Traumatic brain injury

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