Toll-like Receptor 4 Mediates Histone Subunit H3-induced Endothelial Dysfunction in Human Lung Endothelium.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology(2022)

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摘要
Altogether, our results demonstrate that TLR4 signaling is critically involved in mediating histone H3-induced endothelial barrier disruption and inflammation.
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