Fibroblast Senescence: A Risk Factor for Remodeling, Inflammation, and Arrhythmias in the Post-MI Heart.

Elif Sengun,Brett Baggett,Nilufer Turan,Eric Mi,Bianca Kun,John Sedivy,Gideon Koren

FASEB journal : official publication of the Federation of American Societies for Experimental Biology（2022）

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摘要

A significant increase and persistence of CMF senescence likely contributes to pathological tissue remodeling during the post-MI wound healing process. This is likely due to SASP-induced pro-arrhythmogenic chronic inflammation or via direct cell-cell interactions between senescent CMFs and myocytes.

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