Erratum: Localized inhibition of protein phosphatase 1 by NUAK1 promotes spliceosome activity and reveals a MYC-sensitive feedback control of transcription (Molecular Cell (2020) 77(6) (1322–1339.e11), (S1097276520300083), (10.1016/j.molcel.2020.01.008))

(Molecular Cell 77, 1322–1339.e1–e11; March 19, 2020) After discussion among the co-authors, who are all in agreement, Amit Kumar’s contribution to this paper warranted inclusion as a co-author. The authors regret this omission. The paper has since been corrected online, including the author list, acknowledgments, and author contributions, which also appear here. We acknowledge Andreas Schlosser at the mass spectrometry technology platform at Rudolf-Virchow-Zentrum, Würzburg, for the FLAG-NUAK1 and the label-free proteomic experiments. We thank Ulrike Samfass for technical support. This work was supported by grants to M.E. from the European Research Council (AuroMYC), the Federal Ministry of Education and Research (DKTK Therapeutic Targeting of MYC), and the German Research Foundation via the Research Group FOR2314 (Therapeutic Windows) and via grant SFB1309 (to Y.-C.C. and B.K.). G.C. performed most experiments. I.R. and G.C. performed PLAs. C.S.-V. prepared samples for SILAC mass spectrometry. G.M., A.K., S.K., and R.S.V. processed and analyzed SILAC mass spectrometry. Y.-C.C. performed and analyzed TMT mass spectrometry. F.P., L.W., and D.M. designed, developed, and tested the specificity of the BAY-880 compound. A.B. and C.P.A. sequenced ChIP-seq, ChIP-RX, and RNA-seq libraries. S.W. and A.B. analyzed ChIP-seq and RNA-seq data. G.C., S.K., E.W., C.G., R.L., B.K., and M.E. devised, supervised, and interpreted experiments. G.C. and M.E. wrote the paper. Localized Inhibition of Protein Phosphatase 1 by NUAK1 Promotes Spliceosome Activity and Reveals a MYC-Sensitive Feedback Control of TranscriptionCossa et al.Molecular CellJanuary 31, 2020In BriefTumors displaying high MYC levels depend on the NUAK1 kinase. Cossa et al. show that NUAK1 binds protein phosphatase 1 (PP1) and its regulatory subunit PNUTS. They show that this complex associates with the splicing machinery and provides a feedback control of transcription that can be overridden by deregulated MYC. Full-Text PDF Open Access