Leptin accelerates B cell dysfunctions via activating JAK/STAT3/5 and ERK1/2 pathways in patients with systemic lupus erythematosus.

Clinical and experimental rheumatology（2022）

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摘要

Leptin may be a key factor leading to B cell dysfunction by activating JAK/STAT3/5 and ERK1/2 signalling pathways in SLE.

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关键词

leptin, B cells, JAK, STAT3, 5, ERK1, 2, systemic lupus erythematosus, Systemic lupus erythematosus (SLE)

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