Modulation Of Cardiac Extracellular Matrix By A Sustained-Release Synthetic Prostacyclin Agonist Promotes Reverse Remodeling In Hypertrophic Hearts By Inhibiting Fibroblast-To-Myofibroblast Transition In A Mouse Pressure Overload Model
CIRCULATION(2017)
关键词
Prostacyclin, Remodeling, Cardiac hypertrophy, Extracellular matrix, Fibrosis
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