CORM-3 exerts a neuroprotective effect in a rodent model of traumatic brain injury via the bidirectional gut–brain interactions

Li-Min Zhang,Dong-Xue Zhang,Wei-Chao Zheng,Jin-Shu Hu,Lan Fu,Yan Li,Yue Xin,Xu-Peng Wang

Experimental Neurology（2021）

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摘要

•TBI plus HSR caused increased pyroptosis in the vmPFC and jejunum in the acute stage.•TBI plus HSR caused degeneration in morphopathological changes in the jejunum and vmPFC in the late stage.•CORM-3 via intestinal injection provided protection against TBI plus HSR via a bidirectional gut–brain communication.

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关键词

Carbon monoxide,Neuroprotection,Pyroptosis,Jejunum,Traumatic brain injury

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