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Modeling UBQLN2-mediated Neurodegenerative Disease in Mice: Shared and Divergent Properties of Wild Type and Mutant UBQLN2 in Phase Separation, Subcellular Localization, Altered Proteostasis Pathways, and Selective Cytotoxicity

Neurobiology of Disease(2020)

Cited 22|Views61
Key words
Proteostasis,Ubiquilin,Ubiquitin proteasome system,Amyotrophic lateral sclerosis,Frontotemporal dementia,Protein aggregation,Neurodegeneration
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