CDK4/CDK6 inhibition as a novel strategy to suppress the growth and survival of BCR-ABL1 T315I + clones in TKI-resistant CML.

EBioMedicine(2019)

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摘要
This project was supported by the Austrian Science Funds (FWF) projects F4701-B20, F4704-B20 and P30625.
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关键词
CML,BCR-ABL1 mutations,TKI resistance,Hydroxyurea,CDK4/CDK6 – Palbociclib
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