Decreased Vascular Nitric Oxide Production Due to S100A1 Deficiency Preserves Cardiovascular Function and Survival in a Murine Model of Polymycrobial Sepsis
Circulation(2016)
摘要
Introduction: Sepsis-induced myocardial depression is a key component contributing to septic shock. The mechanisms responsible for myocardial dysfunction in sepsis remain undefined. S100A1 a member...
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关键词
Nitric oxide,Blood pressure,Cardiovascular disease,Heart failure
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