Hyperglycaemia augments lipopolysaccharide-induced reduction in rat and human macrophage phagocytosis via the endoplasmic stress-C/EBP homologous protein pathway.

J Iida,S Ishii,Y Nakajima,D I Sessler, H Teramae,K Kageyama, S Maeda, N Anada,M Shibasaki,T Sawa,Y Nakayama

British Journal of Anaesthesia（2019）

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摘要

These findings imply that controlling endoplasmic reticulum stress might provide new strategies for restoring reduced macrophage phagocytosis in sepsis-induced hyperglycaemia.

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关键词

endoplasmic reticulum,hyperglycaemia,macrophage,phagocytosis,resolvin,sepsis

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