A synthetic epoxyeicosatrienoic acid analog prevents the initiation of ischemic acute kidney injury.

Uwe Hoff,Gordana Bubalo,Mandy Fechner,Maximilian Blum,Ye Zhu,Andreas Pohlmann,Jan Hentschel,Karen Arakelyan,Erdmann Seeliger,Bert Flemming,Dennis Gürgen,Michael Rothe,Thoralf Niendorf,Vijaya L Manthati,John R Falck,Michael Haase,Wolf-Hagen Schunck,Duska Dragun

Acta physiologica (Oxford, England)（2019）

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摘要

Pharmacological interventions targeting the CYP-eicosanoid pathway could offer promising new options for AKI prevention. Individual differences in CYP-eicosanoid formation may contribute to the risk of developing AKI in clinical settings. This article is protected by copyright. All rights reserved.

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关键词

Acute kidney injury,CYP-eicosanoids,Inflammation,Reoxygenation,Signalling

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