Inhibition of Apoptosis Signal-Regulating Kinase 1 Attenuates Myocyte Hypertrophy and Fibroblast Collagen Synthesis.
Heart, Lung and Circulation(2019)
摘要
Selective ASK1 inhibition confers anti-hypertrophic and anti-fibrotic effects in cardiac cells, and anti-inflammation in monocytic cells. ASK1 inhibitors may represent novel therapeutic agents to alleviate cardiac remodelling post cardiac injury where hypertrophy, fibrosis and inflammation play critical roles.
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关键词
Apoptosis signal-regulating kinase 1,ASK1 inhibitors,Cardiac hypertrophy,Collagen synthesis,Cardiac remodelling
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