ATF3-dependent cross-talk between cardiomyocytes and macrophages promotes cardiac maladaptive remodeling

Lilach Koren,Dror Alishekevitz,Ofer Elhanani,Alexsander Nevelsky,Tsonwin Hai,Izhak Kehat,Yuval Shaked,Ami Aronheim

International Journal of Cardiology（2015）

引用 33|浏览13

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摘要

Collectively, this study places ATF3 as a key regulator in promoting pressure overload induced cardiac hypertrophy through a cross-talk between cardiomyocytes and macrophages. Inhibiting this cross-talk may serve as a useful approach to blunt maladaptive remodeling processes in the heart.

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关键词

Pressure overload,Cardiac remodeling,Macrophages,Bone marrow transplantation,Cardiac hypertrophy

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