Unique role for ATG5 in neutrophil-mediated immunopathology during M. tuberculosis infection

Jacqueline M. Kimmey,Jeremy P. Huynh,Leslie A. Weiss,Sunmin Park,Amal Kambal,Jayanta Debnath,Herbert W. Virgin,Christina L. Stallings

NATURE（2015）

引用 353|浏览21

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摘要

Genetic engineering in mice reveals that autophagy is not an essential mechanism in myeloid cells for controlling Mycobacterium tuberculosis infection, and that autophagy factor ATG5 protects organisms by regulating neutrophil influx and tissue damage.

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