IL-32γ overexpression accelerates streptozotocin (STZ)-induced type 1 diabetes.

Hyunjhung Jhun,Jida Choi,Jaewoo Hong,Siyoung Lee,Areum Kwak,Eunsom Kim,Seunghyun Jo,Soyoon Ryoo,Yoojung Lim,Do-Young Yoon,Jin Tae Hong,Tae Sung Kim,Youngmin Lee,Keeho Song,Soohyun Kim

Cytokine（2014）

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摘要

•Mouse model of STZ-induced type 1 diabetes was aggravated in IL-32γ TG mice.•Spontaneous elevation of inflammatory cytokines in pancreas of IL-32γ TG mice.•STZ treatment increased TNFα, IFNγ, IL-1β and IL-6 products in pancreas.•IL-32γ/inflammatory cytokines axis contributes to inflammation in STZ-induced diabetes.

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关键词

IL-32γ transgenic mice,Inflammatory cytokine,Streptozotocin (STZ)-induced type I diabetes,Pancreas,Glucose tolerance test

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