Possible involvement of PI3K-dependent pathways in the increased VEGF120 release from osteoblastic cells preloaded with palmitate in vitro.

Rie Moriya,Kazuto Takahashi,Atsuko Kitahara,Hirohisa Onuma,Keiko Handa,Yoshikazu Sumitani,Toshiaki Tanaka,Hidenori Katsuta,Susumu Nishida,Eiji Itagaki,Kouichi Inukai,Hitoshi Ishida

Biochemical and Biophysical Research Communications（2014）

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摘要

•The palmitate-preloaded cells showed the significant increase of VEGF120 release.•The palmitate-induced increase of VEGF120 release from UMR-106 cells may be mediated through, at least partly, TLR4.•The palmitate may increase the expression and secretion of VEGF120 through, mainly, the PI3K pathways in UMR-106 cells.

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关键词

VEGF120,MCP-1,Osteoblast

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