Comment on "Type 1 diabetes in BioBreeding rats is critically linked to an imbalance between Th17 and regulatory T cells and an altered TCR repertoire".

Jan-Luuk Hillebrands,Jan Rozing,Jeroen T J Visser

JOURNAL OF IMMUNOLOGY（2011）

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摘要

In their recent paper, van den Brandt et al. ([1][1]) conclude that diabetes development in diabetes-prone BioBreeding (DP-BB) rats is due to an imbalance between Th17 effector (Th17eff) and regulatory T cells (Tregs) in favor of the Th17eff cells. DP-BB Treg function by itself was not hampered,

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Comment on &quot;Type 1 diabetes in BioBreeding rats is critically linked to an imbalance between Th17 and regulatory T cells and an altered TCR repertoire&quot;.

Comment on "Type 1 diabetes in BioBreeding rats is critically linked to an imbalance between Th17 and regulatory T cells and an altered TCR repertoire".