MBL and C1q compete for interaction with human endothelial cells.

Melinda Oroszlán,Mohamed R Daha,László Cervenak,Zoltán Prohászka,George Füst,Anja Roos

Molecular Immunology（2007）

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摘要

C1q, the recognition molecule of the classical pathway of complement, binds to endothelial cells, leading to cell activation. Mannose-binding lectin (MBL), a recognition molecule of the lectin pathway, is structurally and functionally related to C1q. Therefore, we investigated the interaction of MBL with human umbilical vein endothelial cells (HUVEC).

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关键词

MBL,C1q,Endothelial cell,Clearance,Inflammation,Innate immunity

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