Lack of correlation of onset of lymphomas and levels of murine leukaemia virus in (AKR × CBA/H-T6Crc) F1

There is some evidence that the incidence of lymphomas in AKR hybrids parallels levels of murine leukaemia virus (MuLV). We originally noted that tumours were much delayed in the (AKR × CBA/H-T6Crc) F1 in spite of high levels of MuLV. The study of this cross has been extended to investigate any association between the level of MuLV with the incidence of lymphomas, any possible influence of maternal factors, and also to learn whether the delay of onset of lymphoma in the F1 is associated with the lack of anti-MuLV antibody activity.