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1) Aspirin-like drugs elicit their therapeutic effects through suppression of biosynthesis of prostaglandins (PGs), which exert a wide variety of actions by acting on the PG receptors on the neighboring cells (Fig.). The final output of PG actions has been shown to depend on both intracellular and extracellular circumstances, and their exact molecular mechanisms remain elusive. To clarify these points, we examine the effect of PG receptor deficiency on several processes and therein molecular events in the nervous, immune and reproductive systems.
2) Identification of tissue-specific molecular function of mast cells and its application to drug discovery and prevention of immune diseases.
3) Regulation mechanism of esophageal squamous cancer by micro RNAs (miRs) and its application to cancer therapy.
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INTERNATIONAL JOURNAL OF MOLECULAR SCIENCESno. 3 (2024)
Yuya Tsurudome,Yuya Yoshida,Kengo Hamamura,Takashi Ogino,Sai Yasukochi,Shinobu Yasuo, Ayaka Iwamoto, Tatsuya Yoshihara,Tomoaki Inazumi,Soken Tsuchiya,Toru Takeo,Naomi Nakagata,
International Journal of Molecular Sciencesno. 3 (2024): 1841
IUPHAR/BPS guide to pharmacology CITEno. 1 (2023)
BRITISH JOURNAL OF PHARMACOLOGY (2023): S23-S144
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Miho Hashimoto, Nagisa Makino,Tomoaki Inazumi, Rina Yoshida, Toshiko Sugimoto,Soken Tsuchiya,Yukihiko Sugimoto
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