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The Hagg lab investigates how novel neurotrophic mechanisms and endogenous stemcell might be used to develop CNS repair strategies. We are currently identifying molecular signaling mechanisms and testing small molecules that activate neuroprotective mechanisms might be used to reduce tissue loss after an ischemic stroke injury. We focus on astroglial and endothelial/microvascular mechanisms that protect as well as those that lead to detrimental inflammation in the injured brain regions. Secondly we are identifying molecular regulators of endogenous neural precursors in adult rodents which can be targeted for enhancing neurogenesis and redirect neuroblast migration towards stroke injuries. The long term goal of these studies is to provide information that would lead to better treatment strageties for a variety of human neurological disorders, including spinal cord injury and stroke. Techniques used routinely include various refined microsurgical procedures in the brain and spinal cord of adult rats and mice, pharmacological treatments, regular and confocal immunohistochemistry, Western blotting and real-time PCR.
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Neurobiology of Stress (2024): 100621-100621
CHEMICAL SENSES (2023)
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SSRN Electronic Journal (2021)
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