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For over 20 years we have focused on the role of rearrangements of large segments of the genome, i.e. chromosome translocations, duplications, deletions and inversions, in the common cancers such as breast cancer (reviewed in Edwards, J Pathol 2010;220:244-54). These have proved particularly difficult to analyse in the common cancers, but they are abundant and have powerful effects, particularly where they create fusion genes—genes formed by fusion of two genes, like the BCR-ABL fusion of chronic myeloid leukaemia. We estimated from transcriptome data that an average breast cancer expresses 10 fusion genes (Edwards & Howarth. Breast Cancer Research 2012;14, 303).
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Bernardo Rodriguez-Martin,Eva G. Alvarez,Adrian Baez-Ortega, Jorge Zamora,Fran Supek,Jonas Demeulemeester,Martin Santamarina,Young Seok Ju,Javier Temes,Daniel Garcia-Souto, Harald Detering,Yilong Li,
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crossref(2023)
Nature communicationsno. 1 (2023): 4239-16
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C. J. Peters, Y. Ang, F. D. Ciccarelli, H. Coles, H. G. Coleman, G. Contino, T. Crosby, G. Devonshire, M. Eldridge, A. Freeman, N. Grehan, M. Mccord,
NATURE MEDICINEpp.1-3, (2023)
Huai-En Huang,Suet-Feung Chin,Christophe Ginestier, Valérie-Jeanne Bardou,José Adélaïde, N. Gopalakrishna Iyer,Maria J. Garcia, Jessica C. Pole,Grace M. Callagy, Stephen M. Hewitt, William J. Gullick, Jocelyne Jacquemier,
crossref(2023)
Angela Santonja,Wendy N. Cooper,Matthew D. Eldridge,Paul A. W. Edwards,James A. Morris, Abigail R. Edwards, Hui Zhao,Katrin Heider,Dominique-Laurent Couturier,Aadhitthya Vijayaraghavan,Paulius Mennea, Emma-Jane Ditter,
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