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The papillomaviruses are a large group of pathogens which infect skin and mucous membranes where they induce a spectrum of proliferative disorders from warts to cancer. A subgroup of human papillomavirus (HPV) types, particularly HPV 16 and 18 and their relatives, is closely associated with the development of certain human cancers, most notably cervical cancer. This raises the possibility that intervention in the natural history of HPV infection in the genital tract by prevention or treatment could have a major impact on this cancer, the second commonest malignancy in women in the world.
The ways in which the cell may control the life cycle of the virus and conversely how the virus may exert effects on the growth and differentiation of the host cell are still tantalising mysteries. The contribution of the immune response to papillomavirus infection can greatly affect the degree and duration of disease. Understanding the ways in which the immune system develops a response to the human papillomavirus is important if effective vaccines against viral-associated disease are to be produced.
The ways in which the cell may control the life cycle of the virus and conversely how the virus may exert effects on the growth and differentiation of the host cell are still tantalising mysteries. The contribution of the immune response to papillomavirus infection can greatly affect the degree and duration of disease. Understanding the ways in which the immune system develops a response to the human papillomavirus is important if effective vaccines against viral-associated disease are to be produced.
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