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We are also active in the field of environmental epigenomics, investigating how environmental exposures early in life reprogram the epigenome to determine disease susceptibility across the life-course. In studying how early life exposures to endocrine disrupting compounds (EDCs) influence the development of adult disease, we found that early life EDC exposure can increase susceptibility to NAFLD and liver cancer (Trevino et. al. Nature Communications 2020, Katz et. al. Environmental Health Perspectives 2020). More recently, we have used epigenomic profiling (ChIP-seq) to profile how these exposures induce developmental reprogramming of the epigenome (reviewed in Walker Mol. Endo 2016). Current studies our lab in this area are being conducted as part of the TaRGET II U01 Consortium for NIEHS, which has the goal of identifying epigenomic signatures in target and surrogate tissues that can be developed as biomarkers of exposure and/or risk for environmental causes of disease (Wang et. al. Nat. Biotech 2018).
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论文共 376 篇作者统计合作学者相似作者
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bioRxiv : the preprint server for biology (2024)
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Frontiers in Nutrition (2023): 1146804
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