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My program of research focuses on expanding information obtained from electrocardiographic (ECG) recordings to improve clinical decision-making and patient outcomes in hospital and pre-hospital settings. I helped develop a monitoring strategy, “reduced lead set” technology, a method to derive a multi-lead electrocardiogram from a reduced number of electrodes. I was invited to present my research in reduced lead set technology at the Einthoven Foundation’s Celebration of 100 years of the Electrocardiogram in the Netherlands in 2002. Einthoven won a Nobel Prize for inventing the ECG machine in 1902, and this celebration acknowledged scientists who had made important and lasting contributions to the field.
Results of studies from my research laboratory (ECG Monitoring Research Lab in the School of Nursing) have informed the development of ST-segment (ischemia) monitoring algorithms for cardiac monitors. We discovered causes of false ischemia monitoring alarms, such as changes in a patient’s body position, which can alter waveforms and mimic the electrocardiographic changes of myocardial ischemia. Our publications provided evidence for the importance of ischemia monitoring in patients presenting to the emergency room with chest pain or treated in hospital units for acute coronary syndromes. A series of studies from my laboratory have shown that transient ischemia following treatment for acute coronary syndromes is: (1) usually missed with routine “arrhythmia” monitoring leads, (2) associated with poor hospital outcomes and, (3) most often (80%) clinically silent (asymptomatic). In addition, we were the first to publish that automated ST segment ischemia monitoring provided prognostic information above and beyond the initial standard 12-lead ECG in patients with acute coronary syndrome.
Results of studies from my research laboratory (ECG Monitoring Research Lab in the School of Nursing) have informed the development of ST-segment (ischemia) monitoring algorithms for cardiac monitors. We discovered causes of false ischemia monitoring alarms, such as changes in a patient’s body position, which can alter waveforms and mimic the electrocardiographic changes of myocardial ischemia. Our publications provided evidence for the importance of ischemia monitoring in patients presenting to the emergency room with chest pain or treated in hospital units for acute coronary syndromes. A series of studies from my laboratory have shown that transient ischemia following treatment for acute coronary syndromes is: (1) usually missed with routine “arrhythmia” monitoring leads, (2) associated with poor hospital outcomes and, (3) most often (80%) clinically silent (asymptomatic). In addition, we were the first to publish that automated ST segment ischemia monitoring provided prognostic information above and beyond the initial standard 12-lead ECG in patients with acute coronary syndrome.
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